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At low thiamine concentrations, such as those normally found in the human body, this transfer is achieved by a specific thiamine transporter molecule that requires energy

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(1997) demonstrated that perinatal exposure to 10 mg/ml Mn in drinking water induced hyperactivity in rats in association with cerebral cortex thickening, although no significant alterations in TH activity, dopamine, and GFAP levels were observed (Pappas et al., 1997), suggesting involvement of other potential mechanisms in addition to dopamine