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Despite the presence of localized epithelial lesions, freeze-fracture electron microscopy reveals that discontinuous TJ strands contribute to the dysfunction of the intestinal barrier

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[281] introduced exendin-4 (Ex-4), a negatively charged analog of glucagon-like peptide-1, into the open lumen of HNTs, which were subsequently blended with PCL/CA spinning solution to fabricate nanofiber scaffolds for tendon regeneration