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Upon activation, TAK1 phosphorylates the downstream inhibitory protein IκBα, leading to dissociation of NF-κBp65 from IκBα and subsequent translocation into the nucleus for transcriptional regulation of related genes

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Safety and effectiveness has not been established for patients with a neurological disease other than Parkinson’s disease or essential tremor, previous surgical ablation procedures, dementia, coagulopathies, or moderate to severe depression