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Disease-specific shifts in population abundance highlight the context-dependent plasticity of human microglia, with a glycoprotein non-metastatic melanoma protein B (GPNMB)-high microglial subtype notably expanded in both AD and multiple sclerosis, implicating this population in neurodegenerative inflammatory cascades ( Figure 13 )

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Since RP is a genetic disorder primarily caused by mutations, fetuses carrying causative mutations could theoretically be identified through genetic testing during pregnancy, such as Non-Invasive Prenatal Testing (NIPT) or amniocentesis