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The transcription factor NF- ฮบ B is central to the initiation and regulation of PRR-mediated inflammatory responses (Liu et al., 2017), and the impact of dopamine on NF- ฮบ B has been examined in a variety of immune cell subsets (Takeuchi and Fukunaga, 2003

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One hypothesis from researchers at Harvard Medical School: chronic NNMT suppression might alter methylation donor availability, since NNMT consumes S-adenosylmethionine (SAM) as a methyl donor

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