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It is evident that oxidative stress is a major driving mechanism in COPD and may account for many of the pathophysiological changes that occur [20]

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Constitutive AR signaling in PCA cells despite androgen deprivation Multiple investigations have demonstrated the importance of AR signaling in CRPC cells, where constitutively high nuclear AR protein levels were observed in both CRPC cell lines grown in the absence of hormone stimulation [72] and in both tumor samples from patients undergoing ADT [68] and in circulating tumor cells (CTC) from CRPC patients [73]