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Oxalate lowering via AGXT overexpression ameliorates MASH Considering that AGXT is consistently downregulated in MASH leading to hepatic oxalate overproduction in association with disease severity, and that the loss of AGXT accelerates MASH progression in mice 12 , we next sought to determine whether overexpression of AGXT in hepatocytes would improve MASH outcomes by lowering oxalate production

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Treating SC bioavailability as equivalent to IV