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In microglia, α-Syn fibrils and DA neuron degeneration work together to activate the NLRP3 inflammasome, and inhibition of NLRP3 with MCC950 dramatically decreases the quantity of α-Syn aggregates in the substantia nigra (SN) of mice, preventing the development of motor impairments and dopaminergic neuron degeneration in the SN [64]

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Abstract Cell fates are controlled by ‘pioneers’, sequence-specific transcription factors (TFs) that bind recognition motifs on nucleosomes (‘pioneer binding’)