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Hyperglycemia produces oxidative stress, upregulating endothelial NOX1/O 2 •− , but not NOX2, mediated reduction in NO bioavailability, eNOS uncoupling and endothelial dysfunction, and generation of pro-inflammatory/pro-fibrotic factors [152]

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[DOI] [PMC free article] [PubMed] [Google Scholar] 459.Uchida-Maruki H., Inagaki H., Ito R., Kurita I., Sai M., Ito T