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The ER stress arising from feeding has been proposed to be driven in part by mTOR and the consequent upregulation of protein synthesis, which would be expected to increase the load of client proteins entering the ER (Ozcan et al., 2008

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This is due to the presence of estrogen receptors (ER) in various tissues and cells, including the uterus, breast, liver, and immune cells, which are regulated by estrogen [165]