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Results of animal studies show an upregulated GALR1 expression in the prefrontal cortex in a postnatal depression model, while 5-HT and 5-HIAA expression were downregulated [194]

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However, APAP did not cause JNK activation in HepaRG cells [28] despite that mitochondrial dysfunction and oxidant stress are central to the mechanism of cell death in this cell type [29]

Glutathione is also a substrate for the glutathione peroxidases and glutathione S