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1 An alternative mechanism by which medications may enhance APAP hepatotoxicity involves competitive utilization of the glucouronidation pathway, resulting in increased metabolism towards the CYP system and thus enhanced formation of hepatotoxic substrates

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This inflammatory modulation does not suppress the necessary early inflammatory response entirely — instead, it appears to regulate its intensity and duration, preventing the chronic, excessive inflammation that delays healing in diabetic wounds, chronic ulcers, and aged skin